SINDROME DE OSGOOD SCHLATTER PDF

Ultrasonography[ edit ] This test can see various warning signs that predict if OSD might occur. Ultrasonography can detect if there is any tissue swelling and cartilage swelling. It has unique features such as detection of an increase of swelling within the tibia or the cartilage surrounding the area and can also see if there is any new bone starting to build up around the tibial tuberosity. Types[ edit ] Three types of avulsion fractures. OSD may result in an avulsion fracture , with the tibial tuberosity separating from the tibia usually remaining connected to a tendon or ligament.

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Ultrasonography[ edit ] This test can see various warning signs that predict if OSD might occur. Ultrasonography can detect if there is any tissue swelling and cartilage swelling. It has unique features such as detection of an increase of swelling within the tibia or the cartilage surrounding the area and can also see if there is any new bone starting to build up around the tibial tuberosity. Types[ edit ] Three types of avulsion fractures. OSD may result in an avulsion fracture , with the tibial tuberosity separating from the tibia usually remaining connected to a tendon or ligament.

This injury is uncommon because there are mechanisms that prevent strong muscles from doing damage. The fracture on the tibial tuberosity can be a complete or incomplete break. Type I: A small fragment is displaced proximally and does not require surgery.

Type II: The articular surface of the tibia remains intact and the fracture occurs at the junction where the secondary center of ossification and the proximal tibial epiphysis come together may or may not require surgery. Type III: Complete fracture through articular surface including high chance of meniscal damage. This type of fracture usually requires surgery. Differential diagnosis[ edit ] Sinding-Larsen and Johansson syndrome , [16] is an analogous condition involving the patellar tendon and the lower margin of the patella bone, instead of the upper margin of the tibia.

Prevention[ edit ] Example of how to stretch the quadriceps muscle. Lack of flexibility in these muscles can be direct risk indicator for OSD. Muscles can shorten, which can cause pain but this is not permanent.

The main stretches for prevention of OSD focus on the hamstrings and quadriceps. However, bracing may give comfort and help reduce pain as it reduces strain on the tibial tubercle.

The knee should be kept straight, legs should be lifted and lowered slowly, and reps should be held for three to five seconds. Rehabilitation focuses on muscle strengthening, gait training, and pain control to restore knee function.

Quadriceps and hamstring exercises prescribed by rehabilitation experts restore flexibility and muscle strength. Education and knowledge on stretches and exercises is important. Exercises should lack pain and increase gradually with intensity. The patient is given strict guidelines on how to perform exercises at home to avoid more injury. This helps to avoid pain, stress, and tight muscles that lead to further injury that oppose healing. Knee orthotics such as patella straps and knee sleeves help decrease force traction and prevent painful tibia contact by restricting unnecessary movement, providing support, and also adding compression to the area of pain.

Prognosis[ edit ] The condition is usually self-limiting and is caused by stress on the patellar tendon that attaches the quadriceps muscle at the front of the thigh to the tibial tuberosity. Following an adolescent growth spurt, repeated stress from contraction of the quadriceps is transmitted through the patellar tendon to the immature tibial tuberosity.

This can cause multiple subacute avulsion fractures along with inflammation of the tendon, leading to excess bone growth in the tuberosity and producing a visible lump which can be very painful, especially when hit.

Activities such as kneeling may also irritate the tendon. Several authors have tried to identify the actual underlying etiology and risk factors that predispose Osgood—Schlatter disease and postulated various theories. However, currently it is widely accepted that Osgood—Schlatter disease is a traction apophysitis of the proximal tibial tubercle at the insertion of the patellar tendon caused by repetitive micro-trauma. In other words, Osgood—Schlatter disease is an overuse injury and closely related to the physical activity of the child.

It was shown that children who actively participate in sports are affected more frequently as compared with non-participants. In some cases the symptoms do not resolve until the patient is fully grown. In addition, in , a case study of patients was observed over 12 to 24 months.

It occurs more frequently in boys than in girls, with reports of a male-to-female ratio ranging from to as high as It has been suggested that difference is related to a greater participation by boys in sports and risk activities than by girls.

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Osgood–Schlatter disease

Abstract The pathology of Osgood-Schlatter occurs in the adolescence in the phase denominated growth pull, has as main characteristics pain in the knee specially to the efforts that need a strong contraction of the muscle quadriceps, and a visible bony proeminence. To the exam of ray-x, an irregular line is observed in the tuberosity tibial, it can be present a significant bony avultion. This issue proposes to verify through a bibliographic review, the physiotherapeutic resources for Prevention and treatment of Osgood Schlatter lesion. The literature review was made through books, scientific articles and databases of Scielo, Lilacs and Aleph.

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